Experiments from flight- and ground-based model systems suggest that unexpected alterations of the human lymphoblastoid cell line Jurkat, as well as effects on cell growth, metabolism, and apoptosis, can occur in altered gravity conditions. Using a desktop random positioning machine (RPM), we investigated the effects of simulated microgravity on Jurkat cells and their multidrug-resistant subline, Jurkat/A4 cells. The viability of Jurkat/A4 cells decreased after simulated microgravity in contrast with the Jurkat cells. At the same time, the viability between the experimental Jurkat cells and control Jurkat cells was not significantly different. Of note, Jurkat cells appeared as less susceptible to apoptosis than their multidrug-resistant clone Jurkat/A4 cells, whereas cell-cycle analysis showed that the percentage of Jurkat/A4 cells in the S-phase was increased after 72 and 96 h of RPM-simulated microgravity relative to their static counterparts. The differences in Jurkat cells at all phases between static and simulated microgravity were not significant. The surface expression of the intercellular adhesion molecule 3 (ICAM-3)-also known as cluster of differentiation (CD)50-protein was changed for Jurkat/A4 cells following exposure to the RPM. Changes in cell morphology were observed in the Jurkat/A4 cells after 96 h of RPM-simulated microgravity. Thus, we concluded that Jurkat/A4 cells are more sensitive to RPM-simulated microgravity as compared with the parental Jurkat cell line. We also suggest that intercellular adhesion molecule 3 may be an important adhesion molecule involved in the induction of leukocyte apoptosis. The Jurkat/A4 cells with an acquired multidrug resistance phenotype could be a useful model for studying the effects of simulated microgravity and testing anticancer drugs.

译文

:基于飞行和地面的模型系统的实验表明,人的淋巴母细胞样细胞系Jurkat发生意想不到的改变,以及对细胞生长,代谢和细胞凋亡的影响,都可能在重力条件改变的情况下发生。我们使用台式随机定位机(RPM),研究了模拟微重力对Jurkat细胞及其耐多药亚系Jurkat / A4细胞的影响。与Jurkat细胞相比,模拟微重力作用后Jurkat / A4细胞的活力降低。同时,实验Jurkat细胞和对照Jurkat细胞之间的生存力没有显着差异。值得注意的是,Jurkat细胞似乎比耐多药的克隆Jurkat / A4细胞更不容易凋亡,而细胞周期分析显示,RPM分别在72和96 h后,S期的Jurkat / A4细胞百分比增加了-模拟相对于其静态对应物的微重力。静态和模拟微重力之间所有阶段的Jurkat细胞差异均不显着。暴露于RPM后,Jurkat / A4细胞的细胞间粘附分子3(ICAM-3)(也称为分化簇(CD)50-蛋白)的表面表达发生了变化。 RPM模拟微重力96 h后,在Jurkat / A4细胞中观察到细胞形态的变化。因此,我们得出的结论是,与亲本Jurkat细胞系相比,Jurkat / A4细胞对RPM模拟的微重力更敏感。我们还建议细胞间粘附分子3可能是参与白细胞凋亡诱导的重要粘附分子。具有获得的多药耐药表型的Jurkat / A4细胞可能是研究模拟微重力作用和测试抗癌药物的有用模型。

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