Three types of overlap occur among the disease states chronic fatigue syndrome (CFS), fibromyalgia (FM), multiple chemical sensitivity (MCS) and posttraumatic stress disorder (PTSD). They share common symptoms. Many patients meet the criteria for diagnosis for two or more of these disorders and each disorder appears to be often induced by a relatively short-term stress which is followed by a chronic pathology, suggesting that the stress may act by inducing a self-perpetuating vicious cycle. Such a vicious cycle mechanism has been proposed to explain the etiology of CFS and MCS, based on elevated levels of nitric oxide and its potent oxidant product, peroxynitrite. Six positive feedback loops were proposed to act such that when peroxynitrite levels are elevated, they may remain elevated. The biochemistry involved is not highly tissue-specific, so that variation in symptoms may be explained by a variation in nitric oxide/peroxynitrite tissue distribution. The evidence for the same biochemical mechanism in the etiology of PTSD and FM is discussed here, and while less extensive than in the case of CFS and MCS, it is nevertheless suggestive. Evidence supporting the role of elevated nitric oxide/peroxynitrite in these four disease states is summarized, including induction of nitric oxide by common apparent inducers of these disease states, markers of elevated nitric oxide/peroxynitrite in patients and evidence for an inductive role of elevated nitric oxide in animal models. This theory appears to be the first to provide a mechanistic explanation for the multiple overlaps of these disease states and it also explains the origin of many of their common symptoms and similarity to both Gulf War syndrome and chronic sequelae of carbon monoxide toxicity. This theory suggests multiple studies that should be performed to further test this proposed mechanism. If this mechanism proves central to the etiology of these four conditions, it may also be involved in other conditions of currently obscure etiology and criteria are suggested for identifying such conditions.

译文

疾病状态慢性疲劳综合症(CFS),纤维肌痛(FM),多种化学敏感性(MCS)和创伤后应激障碍(PTSD)之间发生三种类型的重叠。他们有共同的症状。许多患者符合两种或两种以上疾病的诊断标准,每种疾病似乎通常是由相对短期的压力所致,随后是慢性病理,这表明压力可能通过诱导自我延续的恶性行为而起作用。循环。有人提出了这种恶性循环机制来解释CFS和MCS的病因,原因是一氧化氮及其有效氧化剂过氧化亚硝酸盐的含量升高。提出了六个正反馈回路来起作用,使得当过亚硝酸盐水平升高时,它们可能仍保持升高状态。涉及的生物化学不是高度组织特异性的,因此症状的变化可以用一氧化氮/过亚硝酸盐组织分布的变化来解释。此处讨论了PTSD和FM病因学中相同的生化机制的证据,尽管不如CFS和MCS广泛,但仍具有启发性。总结了支持升高一氧化氮/过氧亚硝酸盐在这四种疾病状态中作用的证据,包括这些疾病状态的常见表观诱导剂对一氧化氮的诱导作用,患者中一氧化氮/过氧亚硝酸盐升高的标志物以及升高的一氧化氮的诱导作用的证据。动物模型中的氧化物。该理论似乎是第一个为这些疾病状态的多重重叠提供机械解释的理论,它还解释了它们许多常见症状的起源以及与海湾战争综合症和慢性一氧化碳毒性后遗症的相似性。该理论建议应进行多项研究,以进一步测试这种提议的机制。如果此机制证明对这四种情况的病因至关重要,则它也可能与目前尚不清楚的病因的其他情况有关,并提出了确定此类情况的标准。

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