Baroreflex control of the heart rate is significantly reduced during aging. However, neural mechanisms that underlie such a functional reduction are not fully understood. We injected the tracer DiI into the left nucleus ambiguus (NA), then used confocal microscopy and a Neurolucida Digitization System to examine qualitatively and quantitatively vagal efferent projections to cardiac ganglia of young adult (5-6 months) and aged (24-25 months) rats (Sprague Dawley). Fluoro-Gold was injected intraperitoneally to counterstain cardiac ganglionic principal neurons (PNs). In aged, as in young rats, NA axons projected to all cardiac ganglia and formed numerous basket endings around PNs in the hearts. However, significant structural changes were found in aged rats compared with young rats. Vagal efferent axons contained abnormally swollen axonal segments and exhibited reduced or even absent synaptic-like terminals around PNs, such that the numbers of vagal fibers and basket endings around PNs were substantially reduced (P < 0.01). Furthermore, synaptic-like varicose contacts of vagal cardiac axons with PNs were significantly reduced by approximately 50% (P < 0.01). These findings suggest that vagal efferents continue to maintain homeostatic control over the heart during aging. However, the marked morphological reorganization of vagal efferent axons and terminals in cardiac ganglia may represent the structural substrate for reduced vagal control of the heart rate and attenuated baroreflex function during aging.

译文

衰老期间心率的压力反射控制明显降低。但是,尚不完全了解这种功能降低的神经机制。我们将示踪剂DiI注射到左核歧义 (NA) 中,然后使用共聚焦显微镜和神经清醒数字化系统定性和定量地检查迷走神经传出对年轻成人 (5-6个月) 和年龄 (24-25岁) 大鼠 (Sprague Dawley)。腹膜内注射氟金以反染色心脏神经节主要神经元 (PNs)。在年龄较大的大鼠中,NA轴突投射到所有心脏神经节,并在心脏PNs周围形成许多篮状末端。然而,与年轻大鼠相比,老年大鼠发现了明显的结构变化。迷走神经传出轴突包含异常肿胀的轴突段,并在PNs周围表现出减少或甚至不存在突触样末端,使得PNs周围迷走神经纤维和篮状末端的数量显著减少 (P <0.01)。此外,迷走神经心脏轴突与PNs的突触样静脉曲张接触显着减少了约50% (P <0.01)。这些发现表明,在衰老过程中,迷走神经传出继续保持对心脏的稳态控制。然而,心脏神经节中迷走神经传出轴突和终末的明显形态重组可能代表了衰老过程中迷走神经对心率的控制降低和压力反射功能减弱的结构底物。

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