Purinergic stimulation of human airway epithelia results in a prolonged increase in ciliary beat frequency that depends on calcium-mediated cAMP production [Lieb, T., Wijkstrom Frei, C., Frohock, J.I., Bookman, R.J. and Salathe, M. (2002) Prolonged increase in ciliary beat frequency after short-term purinergic stimulation in human airway epithelial cells. J. Physiol. (Lond.) 538, 633-646]. Here, fully differentiated human airway epithelial cells in culture are shown to express calcium-stimulated transmembrane adenylyl cyclase (tmAC) isoforms (types 1, 3, and 8) by reverse transcription polymerase chain reaction. Immunohistochemistry of tracheal sections and fully differentiated airway epithelial cell cultures revealed polarized expression of these tmACs, with types 1 and 8 localized to the apical membrane and thus at the position required for ciliary regulation. Real-time, ciliated-cell specific cAMP production by tmACs upon apical, purinergic stimulation with UTP was confirmed using fluorescent energy resonance transfer between fluorescently tagged PKA subunits.

译文

:对人气道上皮的脓毒症刺激导致纤毛搏动频率的延长增加,这取决于钙介导的cAMP的产生[Lieb,T.,Wijkstrom Frei,C.,Frohock,J.I.,Bookman,R.J.和Salathe,M.(2002)短期嘌呤能刺激人气道上皮细胞后,纤毛搏动频率延长。 J.生理学。 (Lond。)538,633-646]。在这里,在培养物中完全分化的人气道上皮细胞显示出通过逆转录聚合酶链反应表达钙刺激的跨膜腺苷酸环化酶(tmAC)同工型(1、3和8型)。气管切片和完全分化的气道上皮细胞培养物的免疫组织化学揭示了这些tmAC的极化表达,其中1型和8型位于心尖膜,因此位于睫状调节所需的位置。使用荧光标记的PKA亚基之间的荧光能量共振转移,可以确认tmAC在使用UTP进行根尖,嘌呤能刺激后实时产生纤毛细胞,并产生特定的cAMP。

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