The angiogenic factor vascular endothelial growth factor (VEGF) predicts outcome in primary breast carcinoma. Alteration of the p53 gene causes down-regulation of the expression of thrombospondin-1, a natural inhibitor of angiogenesis. This study was conducted to investigate the association between mutant p53 protein and VEGF expression, and the prognostic value of these factors. VEGF165 and p53 protein were measured in tumour cytosols by enzyme immunoassays. Recurrence-free survival (RFS) and overall survival (OS) were estimated in 833 consecutive patients, 485 node-negative (NNBC) and 348 node-positive (NPBC) with primary invasive breast cancer. A significant association was found between mutant p53 protein and VEGF expression. Univariate analysis showed both p53 and VEGF to be significant predictors of survival. Similar correlation was seen when p53 was combined with VEGF. Univariate analysis of NNBC showed significant prognostic value of p53 for OS, also when combined with VEGF expression; for NPBC, significant reductions in RFS and OS were seen for p53-positive patients, and these findings were enhanced when combined with VEGF, also in the sub-group receiving adjuvant endocrine treatment. Multivariate analysis showed both p53 and VEGF as independent predictors of OS in all groups. When the 2 factors were combined, an increased relative risk of 2.7 was seen for OS in the group with both p53 positivity and high VEGF content, as compared with 1.7 in the group with one risk factor. The results suggest an association between loss of wt-p53 and increased VEGF expression, indicating that angiogenic activity may depend, at least partly, on altered p53-protein function. Combination of these 2 biological markers appears to give additional predictive information of survival. A high-risk group of patients was associated with p53 positivity and higher VEGF content.

译文

血管生成因子血管内皮生长因子 (VEGF) 可预测原发性乳腺癌的预后。p53基因的改变导致血管生成的天然抑制剂thrombospondin-1的表达下调。这项研究旨在研究突变型p53蛋白与VEGF表达之间的关系,以及这些因素的预后价值。通过酶免疫测定法在肿瘤细胞质中测量了VEGF165和p53蛋白。在833位连续患者中估计无复发生存率 (RFS) 和总生存率 (OS),485淋巴结阴性 (NNBC) 和348淋巴结阳性 (NPBC) 原发性浸润性乳腺癌。发现突变型p53蛋白与VEGF表达之间存在显着关联。单因素分析显示p53和VEGF都是生存的重要预测指标。当p53与VEGF结合时,观察到类似的相关性。NNBC的单因素分析显示,p53对OS具有重要的预后价值,同时与VEGF表达相结合; 对于NPBC,p53-positive患者的RFS和OS显著降低,当与VEGF结合时,这些发现增强,同样在接受辅助内分泌治疗的亚组中。多变量分析显示,所有组中p53和VEGF都是OS的独立预测因子。当将这2个因素合并时,在p53阳性和高VEGF含量的组中,OS的相对2.7风险增加,而在具有一个危险因素的组中,与1.7相比。结果表明wt-p53损失与VEGF表达增加之间存在关联,表明血管生成活性可能至少部分取决于p53-protein功能的改变。这两种生物标记的组合似乎提供了生存的其他预测信息。高危组患者与p53阳性和较高的VEGF含量有关。

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