Respiratory failure (RF) developed in 43 (40.2 percent) of 107 patients with acute organophosphate or carbamate poisoning; 22 (51.2 percent) died. The 64 patients who did not develop RF survived. All cases of RF developed within 96 hours after poisoning: within 24 hours in 35 patients (acute onset) and between 24 and 96 hours in eight patients (subacute onset). Severity of poisoning was the primary determinating factor for RF. Cardiovascular collapse and pneumonia were also associated with RF. In 19 patients with cardiovascular collapse, 17 had acute onset of RF and two had subacute onset. In 28 patients with pneumonia, 17 developed acute onset of RF and eight developed subacute onset. No organophosphorus compound caused RF more frequently than another. The duration of ventilator support for subacute RF was significantly longer than for acute RF (287 +/- 186 vs 115 +/- 103 hours, p = 0.02). The use of pralidoxime did not reduce the incidence of RF. We found that severity of poisoning, cardiovascular collapse, and pneumonia were the predisposing factors to RF. The golden time for treatment of acute organophosphate or carbamate poisoning was the initial 96 hours. No RF occurred after this time. Aggressive treatment and prevention of the above three factors will reduce the incidence of RF, or in other words, reduce the mortality.

译文

:107例急性有机磷酸酯或氨基甲酸酯中毒患者中,有43例(40.2%)出现呼吸衰竭(RF); 22人(51.2%)死亡。 64例未发生RF的患者幸存。所有RF病例在中毒后96小时内发展:35例患者在24小时内(急性发作),而8例患者在24到96小时内(亚急性发作)。中毒的严重程度是影响RF的主要因素。心血管衰竭和肺炎也与射频有关。在19例心血管衰竭患者中,有17例急性RF发作,其中2例为亚急性发作。在28例肺炎患者中,有17例发展为RF急性发作,有8例为亚急性发作。没有一种有机磷化合物比另一种引起RF的频率更高。亚急性RF的呼吸机支持时间明显长于急性RF(287 /-186 vs 115 /-103小时,p = 0.02)。使用普利昔肟并不能降低RF的发生率。我们发现中毒的严重程度,心血管衰竭和肺炎是导致RF的诱因。治疗急性有机磷酸酯或氨基甲酸酯中毒的黄金时间是最初的96小时。在此时间之后没有RF发生。积极地治疗和预防以上三个因素将降低RF的发生率,换句话说,可以降低死亡率。

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