Obesity is a chronic metabolic disease caused by a long-term imbalance between energy intake and expenditure. The discovery of three different shades of adipose tissues has implications in terms of understanding the pathogenesis and potential interventions for obesity and its related complications. Fat browning, as well as activation of brown adipocytes and new beige adipocytes differentiated from adipogenic progenitor cells, are emerging as interesting and promising methods to curb obesity because of their unique capacity to upregulate non-shivering thermogenesis. This capacity is due to catabolism of stored energy to generate heat through the best characterized thermogenic effector uncoupling protein 1 (UCP1). A variety of phytochemicals have been shown in the literature to contribute to thermogenesis by acting as chemical uncouplers, UCP1 inducers or regulators of fat differentiation and browning. In this review, we summarize the mechanisms and strategies for targeting adipose-mediated thermogenesis and highlight the role of phytochemicals in targeting adipose thermogenesis to fight against obesity. We also discuss proposed targets for how these phytochemical molecules promote BAT activity, WAT browning and beige cell development, thereby offering novel insights into interventional strategies of how phytochemicals may help prevent and manage obesity via adipose thermogenesis.

译文

肥胖是一种长期的能量摄入和消耗不平衡所导致的慢性代谢性疾病。发现三种不同阴影的脂肪组织对了解肥胖及其相关并发症的发病机理和潜在干预措施具有重要意义。脂肪褐变,以及从脂肪原细胞分化的棕色脂肪细胞和新的米色脂肪细胞的激活,由于其独特的上调非颤抖的产热能力而成为抑制肥胖的有趣且有前途的方法。这种能力是由于储存能量的分解代谢通过最典型的热效应解偶联蛋白1 (UCP1) 产生热量。文献中已显示出多种植物化学物质通过充当化学解耦剂,UCP1诱导剂或脂肪分化和褐变的调节剂来促进产热。在这篇综述中,我们总结了靶向脂肪介导的产热的机制和策略,并强调了植物化学物质在靶向脂肪产热以对抗肥胖中的作用。我们还讨论了这些植物化学分子如何促进BAT活性,WAT褐变和米色细胞发育的拟议目标,从而为植物化学物质如何通过脂肪产热帮助预防和管理肥胖的干预策略提供了新的见解。

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