The purpose of the present study was to determine if our recently introduced novel working rat heart preparation could be used to study wall stress-induced dysrhythmias. A double cannula, which consisted of an outer cannula that, was inserted in the aorta and an inner cannula that was advanced into the left ventricle was used. The perfusion flowed through the inner cannula into the left ventricle and was ejected from there into the aorta. Afterload was changed suddenly from 60 to 160 Hg of pressure by turning a valve so that the fluid was diverted to a column set at a different height. A sudden increase of aortic pressure that lasted for 10 sec caused cardiac ectopic beats. Wall stress-induced dysrhythmias were more sustained during perfusion with low potassium and low magnesium Krebs-Henseleit solution. Bradykinin (1 microg) or epinephrine (10 microg) was injected as a bolus via an in-line injection port placed at the inner cannula. Bradykinin significantly reduced the incidence of ectopic beats and epinephrine increased the incidence of nonsustained runs of VT. This "working" heart preparation is a convenient tool to study wall stress-induced dysrhythmias.

译文

本研究的目的是确定我们最近引入的新型工作大鼠心脏制剂是否可用于研究壁应力引起的节律障碍。使用了一个双套管,该套管由插入主动脉的外部套管和进入左心室的内部套管组成。灌注通过内套管流入左心室,并从那里喷射到主动脉。通过转动阀使后负荷从60至160 Hg的压力突然改变,使得流体被转移到设置在不同高度的塔中。持续10秒的主动脉压力突然升高导致心脏异位搏动。在使用低钾和低镁Krebs-Henseleit溶液灌注期间,壁应力引起的心律不齐更加持续。缓激肽 (1 microg) 或肾上腺素 (10 microg) 通过放置在内部套管上的在线注射端口作为推注注射。缓激肽显着降低了异位搏动的发生率,肾上腺素增加了非持续性VT运行的发生率。这种 “有效” 的心脏准备是研究壁应力引起的心律不齐的便捷工具。

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