Colipase is a key element in lipase-catalyzed dietary lipids hydrolysis. Although devoid of enzymatic activity, colipase promotes pancreatic lipase activity in the physiological intestinal conditions by anchoring the enzyme on the surface of lipid droplets. Polarization modulation infrared reflection absorption spectroscopy combined with Brewster angle microscopy studies was performed on colipase alone and in various lipid environments to obtain a global view of both conformation and orientation and to assess lipid perturbations. We clearly show that colipase fully inserts into a dilaurin monolayer and promotes the formation of lipid/protein domains, whereas in a phospholipid environment its insertion is only partial, limited to the polar head group. In a mixed 70% phosphatidylcholine/30% dilaurin environment, colipase adsorbs to but does not penetrate deeply into the film. It triggers the formation of diglyceride domains under which it would form a rather uniform layer. We also clearly demonstrate that colipase adopts a preferred orientation when dilaurin is present at the interface. In contrast, at a neutral phospholipid interface, the infrared spectra suggest an isotropic orientation of colipase which could explain its incapacity to reverse the inhibitory effects of these lipids on the lipase activity.

译文

大肠菌酶是脂肪酶催化的膳食脂质水解的关键元素。尽管缺乏酶的活性,但colipase通过将酶锚定在脂质滴表面来促进生理肠道条件下的胰脂肪酶活性。偏振调制红外反射吸收光谱结合布鲁斯特角显微镜研究单独对大脂酶和在各种脂质环境中进行,以获得构象和方向的全局视图,并评估脂质扰动。我们清楚地表明,大肠菌酶完全插入到dilaurin单层中并促进脂质/蛋白质结构域的形成,而在磷脂环境中,其插入仅部分,仅限于极性头基团。在混合的70% 磷脂酰胆碱/30% dilaurin环境中,colip酶吸附到膜中,但不深入渗透到膜中。它触发二甘油酯结构域的形成,在该结构域下它将形成相当均匀的层。我们还清楚地证明,当dilaurin存在于界面处时,colipase采用首选的方向。相反,在中性磷脂界面处,红外光谱表明colipase的各向同性取向,这可以解释其无法逆转这些脂质对脂肪酶活性的抑制作用。

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