Melittin, the soluble peptide of bee venom, has been demonstrated to induce lysis of phospholipid liposomes. We have investigated the dependence of the lytic activity of melittin on lipid composition. The lysis of liposomes, measured by following their mass and dimensions when immobilised on a solid substrate, was close to zero when the negatively charged lipids phosphatidyl glycerol or phosphatidyl serine were used as the phospholipid component of the liposome. Whilst there was significant binding of melittin to the liposomes, there was little net change in their diameter with melittin binding reversed upon salt injection. For the zwitterionic phosphatidyl choline the lytic ability of melittin is dependent on the degree of acyl chain unsaturation, with melittin able to induce lysis of liposomes in the liquid crystalline state, whilst those in the gel state showed strong resistance to lysis. By directly measuring the dimensions and mass changes of liposomes on exposure to melittin using Dual Polarisation Interferometry, rather than following the florescence of entrapped dyes we attained further information about the initial stages of melittin binding to liposomes.

译文

蜂毒的可溶性肽:蜂毒肽已被证明可诱导磷脂脂质体的裂解。我们已经研究了蜂毒肽的裂解活性对脂质组成的依赖性。当将带负电荷的脂质磷脂酰甘油或磷脂酰丝氨酸用作脂质体的磷脂成分时,通过跟踪固定在固体基质上时它们的质量和尺寸来测量脂质体的裂解接近于零。尽管蜂毒肽与脂质体具有显着结合,但它们的直径几乎没有净变化,而盐注射后蜂毒素的结合被逆转。对于两性离子磷脂酰胆碱,蜂毒肽的溶解能力取决于酰基链不饱和度,蜂毒肽能够诱导处于液晶状态的脂质体溶解,而处于凝胶状态的那些则显示出强烈的抗溶解性。通过使用双极化干涉测量法直接测量暴露于蜂毒肽时脂质体的尺寸和质量变化,而不是跟随捕获的染料的荧光,我们获得了有关蜂毒肽与脂质体结合的初始阶段的更多信息。

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