Junctional ectopic tachycardia (JET) occurs most frequently after operative repair of congenital heart defects. The mechanism is thought to involve direct trauma to the atrioventricular node and His bundle resulting in an ectopic focus. Several therapeutic methods have been described in the pediatric literature with varying degrees of success and complication rates. Because heart rates may exceed 200 to 300 beats per minute, there may be inadequate time for ventricular filling. Ventricular filling can be further compromised because of the asynchrony between the atria and the ventricles. These factors can lead to significant compromise of cardiovascular function in the postoperative patient. We describe our experience with amiodarone in two patients who developed postoperative JET after repair of congenital heart defects. Dosing regimens and previous experience with amiodarone in patients with JET are reviewed.

译文

先天性心脏缺损的手术修复后,交界性异位心动过速 (JET) 最常见。该机制被认为涉及对房室结及其束的直接创伤,导致异位灶。儿科文献中已经描述了几种治疗方法,其成功率和并发症发生率不同。因为心率可能超过每分钟200到300次,所以心室充盈的时间可能不足。由于心房和心室之间的不同步,可以进一步损害心室充盈。这些因素可能会导致术后患者的心血管功能显着受损。我们描述了两名在先天性心脏缺损修复后出现术后喷射的患者中使用胺碘酮的经验。回顾了JET患者的给药方案和以前使用胺碘酮的经验。

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