The aim of present study was to examine the effects of the ethyl acetate fraction (EAF) from Platonia insignis on lipid peroxidation level, nitrite formation, and superoxide dismutase and catalase activities in rat striatum prior to pilocarpine-induced seizures as well as to explore its anticonvulsant activity in adult rats prior to pentylenetetrazole (PTZ)- and picrotoxin (PIC)-induced seizures. Wistar rats were treated with vehicle, atropine (25mg/kg), EAF (0.1, 1, and 10mg/kg), pilocarpine (400mg/kg, P400 group), PTZ (60 mg/kg, PTZ group), PIC (8 mg/kg, PIC group), atropine+P400, EAF+P400, EAF+PTZ, or EAF+PIC. Significant decreases in number of crossings and rearings were observed in the P400 group. The EAF 10+P400 group also had significant increases in these parameters. In addition, in rats treated with P400, there were significant increases in lipid peroxidation and nitrite levels; however, there were no alterations in SOD and catalase activities. In the EAF 10+P400 group, lipid peroxidation and nitrite levels significantly decreased and SOD and catalase activities significantly increased after pilocarpine-induced seizures. Additionally, effects of the EAF were evaluated in PTZ and PIC models. EAF did not increase the latency to development of convulsions induced with PTZ and PIC at the doses tested. Our findings strongly support the hypothesis that EAF does not have anticonvulsant activity in the different models of epilepsy studied. Our results indicate that in the in vivo model of pilocarpine-induced seizures, EAF has antioxidant activity, but not anticonvulsant properties at the doses tested.

译文

:本研究的目的是检查毛果芸香碱诱发癫痫发作之前,柏拉图徽章的乙酸乙酯级分(EAF)对大鼠纹状体脂质过氧化水平,亚硝酸盐形成以及超氧化物歧化酶和过氧化氢酶活性的影响,并探讨在戊四氮(PTZ)和微毒素(PIC)诱发的癫痫发作之前,其抗惊厥活性在成年大鼠中。 Wistar大鼠接受媒介物,阿托品(25mg / kg),EAF(0.1、1和10mg / kg),毛果芸香碱(400mg / kg,P400组),PTZ(60 mg / kg,PTZ组),PIC(8)治疗mg / kg,PIC组),阿托品P400,EAF P400,EAF PTZ或EAF PIC。在P400组中,观察到的过境和繁殖数量显着减少。 EAF 10 P400组的这些参数也有明显增加。此外,在用P400治疗的大鼠中,脂质过氧化和亚硝酸盐水平显着增加。但是,SOD和过氧化氢酶活性没有变化。在EAF 10 P400组中,毛果芸香碱诱发癫痫发作后脂质过氧化和亚硝酸盐水平显着降低,SOD和过氧化氢酶活性显着提高。此外,在PTZ和PIC模型中评估了EAF的效果。在所测试的剂量下,EAF不会增加PTZ和PIC诱发的惊厥发展的潜伏期。我们的发现强有力地支持了以下假设:EAF在所研究的不同癫痫模型中不具有抗惊厥活性。我们的结果表明,在毛果芸香碱诱发的癫痫发作的体内模型中,EAF具有抗氧化活性,但在所测试的剂量下不具有抗惊厥特性。

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