Staphylococcus aureus (S. aureus) causes persistent biofilm-related infections. Biofilm formation by S. aureus is affected by the culture conditions and is associated with certain genotypic characteristics. Here, we show that glucose and sodium chloride (NaCl) supplementation of culture media, a common practice in studies of biofilms in vitro, influences both biofilm formation by 40 S. aureus clinical isolates (methicillin-resistant and methicillin-sensitive S. aureus) and causes variations in biofilm quantification. Methicillin-resistant strains formed more robust biofilms than methicillin-sensitive strains in tryptic soy broth (TSB). However, glucose supplementation in TSB greatly promoted and stabilized biofilm formation of all strains, while additional NaCl was less efficient in this respect and resulted in significant variation in biofilm measurements. In addition, we observed that the ST239-SCCmec (Staphylococcal Cassette Chromosome mec) type III lineage formed strong biofilms in TSB supplemented with glucose and NaCl. Links between biofilm formation and accessory gene regulator (agr) status, as assessed by δ-toxin production, and with mannitol fermentation were not found. Our results show that TSB supplemented with 1.0% glucose supports robust biofilm production and reproducible quantification of S. aureus biofilm formation in vitro, whereas additional NaCl results in major variations in measurements of biofilm formation.

译文

:金黄色葡萄球菌(S. aureus)引起持续的生物膜相关感染。金黄色葡萄球菌形成的生物膜受培养条件的影响,并与某些基因型特征有关。在这里,我们显示,培养基中葡萄糖和氯化钠(NaCl)的补充是体外生物膜研究的一种常见做法,它会同时影响40株金黄色葡萄球菌临床分离株(耐甲氧西林和对甲氧西林敏感的金黄色葡萄球菌)的生物膜形成。并导致生物膜定量的变化。在胰蛋白酶大豆肉汤(TSB)中,耐甲氧西林的菌株比耐甲氧西林的菌株形成更坚固的生物膜。然而,在TSB中补充葡萄糖极大地促进和稳定了所有菌株的生物膜形成,而额外的NaCl在这方面效率较低,并导致生物膜测量值发生显着变化。此外,我们观察到ST239-SCCmec(葡萄球菌盒式染色体mec)III型谱系在补充了葡萄糖和NaCl的TSB中形成了坚固的生物膜。未发现生物膜形成与辅助基因调节剂(agr)状态之间的联系(通过δ-毒素生产评估)以及与甘露醇发酵之间的联系。我们的结果表明,添加1.0%葡萄糖的TSB支持稳定的生物膜产生和体外金黄色葡萄球菌生物膜形成的可再现定量,而额外的NaCl会导致生物膜形成的测量值发生重大变化。

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